PSYCHOLOGICAL MEDICINE心理医学

PSYCHOLOGICAL MEDICINE(英文缩写 PSYCHOL MED),ISSN 0033-2917,eISSN 1469-8978,中文译名:心理医学 是一本学术期刊。本页汇总该期刊的最新影响因子、分区信息以及最新收录于 PubMed 的文献,帮助您快速了解期刊全貌。

2026 年数据 · 影响因子
5.200
JCR 分区
Q1
CAS 分区
B2
近一年发文量
393
本站 PubMed 收录统计

发文量统计区间:2025-09-28 至 2026-09-28,按本站收录文献的发表日期统计。

ISSN: 0033-2917 · eISSN: 1469-8978 · 缩写: PSYCHOL MED ·中文: 心理医学

期刊介绍

选择期刊介绍栏目

期刊简介

《Psychological Medicine》是精神医学与临床心理学领域的国际权威期刊,聚焦精神障碍的病因、机制、诊断、治疗与预防。内容涵盖流行病学、神经影像、遗传学、心理病理学及干预研究,兼顾基础与临床。读者群主要为精神科医师、临床心理学家、神经科学家及公共卫生研究者,适合发表具有理论深度和临床转化价值的高质量研究。

研究方向

主要方向包括精神障碍的流行病学与危险因素、神经生物学与遗传机制、心理社会干预及药物治疗试验、诊断分类与评估工具、公共卫生与预防策略。论文类型以原创研究、系统综述与荟萃分析为主,也接受方法学创新和简短报告,强调实证数据与临床相关性。

期刊特色

研究取向偏重多学科交叉与纵向设计,重视机制探索和临床意义,统计要求较高。论文通常样本量大、方法严谨,适合有扎实数据基础和理论框架的研究者。对临床医生与心理学家而言,该刊是了解精神医学前沿证据的重要来源,也适合作为跨学科合作的发表平台。

投稿难度

投稿难度较高,对创新性、方法学质量和临床价值均有严格要求。建议在投稿前完善研究设计、充分预注册、确保统计稳健,并清晰阐述理论贡献与实践意义。若被拒稿,可根据审稿意见补充分析或转投同领域专业期刊,不宜仅凭分区判断录用可能性。

历年影响因子趋势

JCR 数据年份影响因子JCR 分区
202110.592Q1
20226.900Q1
20235.900Q1
20245.500Q1
20255.200Q1

PSYCHOLOGICAL MEDICINE 最新收录文献

  1. JCR分区: Q1 CAS分区: B2 影响因子: 5.2

    1. Unravelling sex differences in the genetic architecture of anxiety - CORRIGENDUM.

    作者:
    Jihua Hu, Michelle K Lupton, Enda M Byrne, Nicholas G Martin, David C Whiteman, Catherine M Olsen, Jodi T Thomas, Sarah E Medland, Katrina L Grasby, Brittany L Mitchell
    日期:
    2026-09-25

    该文献暂无摘要。

  2. JCR分区: Q1 CAS分区: B2 影响因子: 5.2

    2. Predictors of trajectories of suicidal thoughts in Texas youth depression and suicide research network youth.

    作者:
    Abu Minhajuddin, Rachel A Walker, Manish Kumar Jha, Lynnel C Goodman, Betsy D Kennard, Joseph C Blader, Eric A Storch, Cesar A Soutullo, Jair C Soares, Sarah M Wakefield, Madhukar H Trivedi
    日期:
    2026-09-24

    Suicidal ideation among youth is a public health crisis, creating a need to identify features that can prognosticate suicide-related outcomes. This study characterized trajectories of suicidal thoughts in youth with depression/suicidality and evaluated features predicting trajectories. Participants in the Texas Youth Depression and Suicide Research Network with self-reported suicidal ideation data available for baseline and ≥2 other time points were included ( = 1,449). Group mixture models were used to identify trajectories of suicidal thoughts at four follow-up assessments during the succeeding 6 months. Random Forest analyses evaluated baseline clinical and sociodemographic variables that predicted trajectory group membership after splitting into training (two-thirds) and validation (one-third) samples. Mean (standard deviation) age was 15.6 (2.7) years, and 72.8% were female. Three trajectories were identified: persistently no (43.3%), persistently low (44.2%), and persistently high (12.4%) suicidal thoughts. In Random Forest analyses, self-reported severity of suicide propensity (including pessimism, helplessness, perceived lack of social support, and despair) plus a clinical interview of suicidal ideation predicted these trajectories. The area under the curve in training and validation samples for predicting persistently no suicidal thoughts was 0.83 and 0.80, respectively, and for persistently high suicidal thoughts was 0.87 and 0.87, respectively. Three distinct trajectories of suicidal thoughts were identified. A combination of baseline self-reported measures of depression, pessimism, helplessness, perceived lack of social support, and despair, along with a clinical interview of suicidal ideation, predicted no or high suicidal thoughts over 6 months with high accuracy, providing a clinically useful predictive model of suicide risk.

  3. JCR分区: Q1 CAS分区: B2 影响因子: 5.2

    3. Acceptability and efficacy of a smartphone intervention informed by interpersonal psychotherapy for perinatal mental health: A randomized controlled trial.

    作者:
    Yuko Toshishige, Takuya Okami, Natsumi Chatani, Shiori Kawasaki, Shinobu Goto, Yuki Yoshida, Yusaku Takahashi, Misaki Shimasaki, Yousuke Miharu, Toshiaki A Furukawa, Hiroko Mizushima, Eizaburo Tanaka, Hiroko Maekawa, Tatsuo Akechi
    日期:
    2026-09-24

    Perinatal depression is a global health concern linked to adverse maternal and neonatal outcomes. Although evidence-based psychological interventions, such as interpersonal psychotherapy (IPT), are effective, women lack access to in-person perinatal care. Smartphone-based interventions may offer scalable support; however, evidence from randomized controlled trials (RCTs) evaluating IPT-informed smartphone interventions remains limited. This study assessed the acceptability and efficacy of an IPT-informed smartphone intervention for perinatal mental well-being. Participants were recruited nationwide through online advertisements and postcards. In this nonblinded RCT, 350 pregnant women were assigned to a fully self-guided smartphone application (intervention) or usual care (control). No minimum depression symptom score was required for participation. Acceptability in the intervention group at 8 weeks after enrolment was indicated by the Japanese version of the Client Satisfaction Questionnaire-8 (CSQ-8J) scores of ≥17. Efficacy was defined as the change in Patient Health Questionnaire-9 (PHQ-9) scores from baseline to 8 weeks following enrolment. Depressive symptoms were assessed at 1 month postpartum. The application achieved acceptability, with 90.2% (157/174) of participants scoring ≥17 on the CSQ-8J. No significant between-group differences in depressive symptoms were observed during pregnancy (standardized mean difference (SMD) = 0.11). At 1 month postpartum, the intervention group exhibited significantly lower depressive symptom scores (SMD = 0.28). The smartphone application was highly acceptable. While no significant differences in depressive symptoms were observed during pregnancy, differences favoring the intervention group were observed at 1 month postpartum. An IPT-informed smartphone intervention may represent a promising approach to perinatal mental health support.

  4. JCR分区: Q1 CAS分区: B2 影响因子: 5.2

    4. Cannabis use is associated with increased risk of violence: A systematic review and meta-analysis.

    作者:
    Giulia Trotta, Victoria Rodriguez, Paolo Marino, Meklit Gurmesa, Edoardo Spinazzola, Zhikun Li, Luis Alameda, Marta Di Forti, Robin Murray, Evangelos Vassos
    日期:
    2026-09-24

    Cannabis use is a known risk factor for adverse mental health outcomes, including psychosis, which is frequently associated with aggression. However, the specific impact of cannabis use on the risk of violence remains unclear. This meta-analysis, conducted according to PRISMA guidelines, synthesized evidence from 63 peer-reviewed studies (N = 265,079) identified via MEDLINE, EMBASE, and PsycINFO searches up to November 2024 to clarify the link between cannabis use and violence, either as a perpetrator or as a victim. Random-effects models revealed that cannabis users are at a significantly higher risk of perpetrating violent acts. This association was observed in both psychiatric patients (OR 2.49, 95% CI 1.72-3.61) and the general population (OR 2.05, 95% CI 1.74-2.41), and remained significant across both longitudinal (OR 1.17, 95% CI 1.07-1.28) and cross-sectional (OR 2.37, 95% CI 1.66-3.40) study designs. There was an especially large effect for violence involving a criminal conviction in both general (OR 3.75, 95% CI 2.54-5.53) and psychiatric (OR 4.21, 95% CI 2.86-6.22) populations. Furthermore, cannabis use was significantly associated with an increased risk of becoming a victim of violence (OR 1.49, 95% CI 1.38-1.60), a risk more pronounced in females and mixed-gender cohorts compared to males. This meta-analysis indicates an increased risk of violence, especially that resulting in a criminal conviction, among cannabis users. It also highlights the potential of cannabis use as a target for interventions to decrease violence, and underscores the need for routine assessment of cannabis use among psychiatric patients.

  5. JCR分区: Q1 CAS分区: B2 影响因子: 5.2

    5. Cerebral perfusion alterations are associated with clinical phenotypes and biotypes across the psychosis spectrum.

    5. 脑灌注改变与精神病谱系中的临床表型和生物型相关
    作者:
    Dung Hoang, Victor Zeng, Jothini Sritharan, Henk J M M Mutsaerts, Jan Petr, Rebekah Trotti, Elena Ivleva, Weiying Dai, Scot Hill, Elliot Gershon, Sarah Keedy, Brett Clementz, Carol Taminga, Godfrey Pearlson, Nicolas Bolo, Matcheri Keshavan, Paulo Lizano
    日期:
    2026-09-24

    Cerebral perfusion abnormalities may contribute to the pathophysiology of psychosis spectrum disorders (PSD), including schizophrenia (SZ), schizoaffective disorder (SAD), and bipolar disorder with psychotic features (BP), yet large-scale investigations remain limited. Pseudo-continuous arterial spin labeling scans were acquired from 2412 SZ, 2278 SAD, 158 BP, 401 neurotypical controls (NC) and processed using ExploreASL and FreeSurfer to quantify resting-state cerebral blood flow (CBF) across total gray matter and 60 brain regions. General linear regression compared CBF between NC and diagnostic groups or biologically informed biotypes derived from neurophysiological clustering. Within patient groups, Spearman correlations assessed associations between cognitive/clinical measures and CBF. Analyses were corrected for multiple comparisons. Compared to NC, patient groups showed significantly lower total gray matter CBF (PSD: Cohen's d=-0.124, p-adjusted=0.037; BP: d=-0.259, p-adjusted=0.004; the most severe biotype, Biotype 1: d=-0.222, p-adjusted=0.006). BP and Biotype 1 exhibited significant hypoperfusion in 25 regions spanning bilateral temporal, occipital, inferior frontal cortices, and subcortical structures (BP d= -0.21 to -0.33; Biotype 1 d=-0.19 to -0.26). No significant CBF-cognition associations were observed. Within PSD, higher total gray matter CBF correlated with greater psychotic symptom severity, while right superior temporal sulcus hyperperfusion was associated with greater psychotic, manic, and depressive symptoms. Distinct perfusion patterns exist in PSD, with widespread hypoperfusion in BP and Biotype 1 and hyperperfusion associated with greater symptom severity, supporting perfusion as a potential biomarker to stratify PSD subtypes.

  6. JCR分区: Q1 CAS分区: B2 影响因子: 5.2

    6. Blunted neurophysiological responses to loss feedback characterize the emotionally vulnerable type of clinical samples with gambling disorder.

    作者:
    Gangliang Zhong, Jingyang Liu, Xin Gao, Qiyun Xue, Chao Xue, Xiyuan Zhang, Yujie Bai, Jing Yue, Lihui Wang, Jiang Du, Min Zhao
    日期:
    2026-09-24

    Gambling disorder (GD) is clinically heterogeneous, with trait impulsivity and negative affect (anxiety/depression) representing key sources of variation. This study investigated whether neurophysiological responses to loss feedback differ across GD subgroups defined by these features and whether subgroup membership moderates the association between neural responses and clinical severity. Electroencephalography was recorded from 120 individuals with GD, typed via k-means clustering of impulsivity, anxiety, and depression scores, and 52 healthy controls (HC) during the Balloon Analogue Risk Task. Event-related potentials, including feedback-related negativity (FRN) and P3, were used to index feedback processing. Two GD subgroups were identified: an 'impulsive type' (high impulsivity, low negative affect) and an 'emotionally vulnerable type' (high impulsivity with severe anxiety/depression), with the latter exhibiting greater gambling severity. Neurophysiologically, the emotionally vulnerable type showed blunted FRN and feedback-P3 amplitudes to loss feedback compared to HC, whereas the impulsive type did not differ from HC. Critically, subgroup membership moderated the association between feedback-P3 and gambling severity; a reduced feedback-P3 predicted higher severity only within the emotionally vulnerable type. These findings identify blunted neural responses to loss feedback as a specific characteristic of the emotionally vulnerable GD type. These findings resolve the inconsistent patterns of loss feedback processing in GD and highlight the potential for personalized interventions targeting this subgroup.

  7. JCR分区: Q1 CAS分区: B2 影响因子: 5.2

    7. Psychological resilience and its genetic determinants: Insights from longitudinal analyses of surgery and trauma cohorts.

    作者:
    Hongxi Wang, Yanan Zhang, Lei Liu, Huazhen Yang, Jiesiwei Luo, Zian Cao, Wenwen Chen, Yu Zeng, Fang Fang, Jie Song, Jin Huang, Qian Li, Huan Song
    日期:
    2026-09-21

    To identify resilience to trauma using longitudinal psychological assessments and uncover its genetic components. This prospective cohort study included 12,946 surgical patients from the China Surgery and Anesthesia Cohort (CSAC) with assessments of depressive symptoms from pre-surgery through 1, 6, and 12 months post-surgery, using the 9-item Patient Health Questionnaire (PHQ-9). Linear mixed-effects models predicted post-surgery PHQ-9 scores; individuals with much lower observed PHQ-9 score than expected (i.e. lowest 25th percentile of residuals) were defined as resilient. We performed genome-wide association study (GWAS) analysis to identify variants associated with psychological resilience. We examined the identified variant in 2,285 patients from the China Severe Trauma Cohort (CSTC) using genotype stratification and additive models. CSAC participants had a mean age of 52.2 years and 58.5% were female. Overall, 2,328 (18.0%) were identified as resilient and exhibited a lower prevalence of psychological symptoms. GWAS identified a lead single-nucleotide polymorphism (rs11766511, = 3.082 × 10) mapped to the gene sidekick cell adhesion molecule 1 (), as a candidate locus implicating neural signaling pathways in stress response and resilience. In CSTC, genotype stratification suggested a lower symptom burden among GG genotype of rs11766511 compared with AA carriers (e.g. Beta = -0.94 [-1.76 to -0.12] for PHQ-9 score). The additive genetic model showed a directionally consistent but non-significant protective pattern of the G allele across psychological symptoms. Longitudinal assessments of depressive symptoms offer a reliable method for measuring psychological resilience. The rs11766511 locus mapped to SDK1 may represent a candidate genetic signal for psychological resilience and warrants confirmation.

  8. JCR分区: Q1 CAS分区: B2 影响因子: 5.2

    8. How should we teach psychiatric formulation? A systematic review of the evidence.

    作者:
    Giulia Ferrari, Gareth Knott, Gareth Owen
    日期:
    2026-09-21

    We conducted a systematic review of literature on teaching psychiatric formulation with the aim of reflecting on the findings and proposing recommendations for enhancing formulation pedagogy within psychiatric education. We found categories of publication on formulation tools, educational strategies, and evaluation of outcomes with little continuity between them. Fourteen themes were identified that helped to understand the gaps in the evidence and the unresolved issues of conceptualization. Our review revealed a striking paradox: despite widespread agreement on the centrality of formulation as a core clinical competency in psychiatry, how to teach it remains fragmented, methodologically weak, and conceptually inconsistent. Based on the findings we propose eight recommendations to aid advance in the research of teaching psychiatric formulation.

  9. JCR分区: Q1 CAS分区: B2 影响因子: 5.2

    9. Psychiatric outcomes after streptococcal upper respiratory tract infection in children.

    作者:
    Danish Hafeez, Katharine Lynch-Kelly, Cameron Watson, Sarah Williford, Mark Edwards, Thomas A Pollak
    日期:
    2026-09-21

    Associations between childhood streptococcal upper respiratory infections and subsequent psychiatric outcomes have been described for over 25 years, although clinical evidence remains limited. This study explored psychiatric outcomes after pediatric streptococcal versus non-streptococcal upper respiratory tract infections. This retrospective cohort study used TriNetX, a global federated health research network of over 150 million deidentified electronic medical records. We compared children aged 3-16 with a diagnosis of acute upper respiratory infection (ICD-10 J00-J06) and a test result (positive or negative) within 2 weeks, propensity matched on 70 baseline variables. Main outcomes were the 1-year incidence of any psychiatric disorder, obsessive-compulsive disorder (OCD), tic disorder, anxiety, and restricted oral intake. Before matching, 464,513 children were streptococcal positive and 1,023,171 children were streptococcal negative. After matching, there were 456,221 children in each cohort (48% female). The streptococcal-positive cohort had a reduced risk of any psychiatric disorder (RR 0.92 [0.90, 0.94]), restricted oral intake (0.81 [0.77, 0.86]), and anxiety disorder (0.94 [0.91, 0.97]) at 1 year. Risk of OCD (RR 0.98 [0.86, 1.12]) and tic disorder (RR 0.95 [0.87, 1.04]) was not significantly different between the two cohorts. This large-scale cohort study found no evidence that exposure to is associated with increased risk of the measured psychiatric outcomes.

  10. JCR分区: Q1 CAS分区: B2 影响因子: 5.2

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