MOLECULAR MEDICINE

MOLECULAR MEDICINE(英文缩写 MOL MED),ISSN 1076-1551,eISSN 1528-3658 是一本学术期刊。本页汇总该期刊的最新影响因子、分区信息以及最新收录于 PubMed 的文献,帮助您快速了解期刊全貌。

指标来源：jcr_cas_ifqb

历年影响因子趋势

MOLECULAR MEDICINE 最新收录文献

1. The CD38 Ectoenzyme Family. Abstracts of a meeting, June 8-10, 2006, Torino, Italy.
   Molecular medicine (Cambridge, Mass.) 2006 IF: 8.30 Q1 PMID: 17590919
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2. Betulinic acid-induced Mcl-1 expression in human melanoma--mode of action and functional significance.
   Edgar Selzer, Christiane Thallinger, Christoph Hoeller, Philipp Oberkleiner, Volker Wacheck, Hubert Pehamberger
   Molecular medicine (Cambridge, Mass.) 2002 IF: 8.30 Q1 PMID: 12606824

   Currently there is no information on the regulation of expression and physiological role of the anti-apoptotic protein Mcl-1 in cells of the melanocytic lineage. This study investigates the regulation…

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3. Adenoviral transfer of mda-7 leads to BAX up-regulation and apoptosis in mesothelioma cells, and is abrogated by over-expression of BCL-XL.
   Xiaobo X Cao, Imran Mohuiddin, Sunil Chada, Abner M Mhashilkar, Mustafa K Ozvaran, David J McConkey
   Molecular medicine (Cambridge, Mass.) 2002 IF: 8.30 Q1 PMID: 12606823

   Malignant pleural mesothelioma (MPM) is unresponsive to conventional therapies. Forced expression of the novel tumor suppressor mda-7 gene in other cell types has resulted in decreased growth and apop…

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4. Effects of free fatty acid on polymerization of islet amyloid polypeptide (IAPP) in vitro and on amyloid fibril formation in cultivated isolated islets of transgenic mice overexpressing human IAPP.
   Zhi Ma, Gunilla T Westermark
   Molecular medicine (Cambridge, Mass.) 2002 IF: 8.30 Q1 PMID: 12606822

   Islet amyloid polypeptide (IAPP) is deposited as amyloid in the islets of Langerhans in type 2 diabetes. The mechanism behind the formation of the cytotoxic fibrils is unknown. Islet amyloid develops …

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5. Farnesyl protein transferase inhibition interferes with activation of MAP kinase family members in human peripheral blood monocytes.
   Walter L Vervenne, Carina L Bos, Linda S Rens, Maikel P Peppelenbosch, Dick J Richel
   Molecular medicine (Cambridge, Mass.) 2002 IF: 8.30 Q1 PMID: 12606821

   Farnesyl protein transferase inhibitors have emerged as promising novel agents for combating cancerous disease. Nevertheless, the importance for farnesyl protein transferase enzymatic activity for cel…

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6. Modulation of trophoblast cell death by oxygen and EGF.
   Jonathan Perkins, Justin St John, Asif Ahmed
   Molecular medicine (Cambridge, Mass.) 2002 IF: 8.30 Q1 PMID: 12606820

   Preeclampsia, a maternal hypertensive disease, is characterized by shallow invasion of the maternal spiral arterioles resulting in hypoxia/reperfusion type insult; however, the molecular mechanism is …

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7. Peripheral benzodiazepine binding sites in platelets of patients affected by mitochondrial diseases and large scale mitochondrial DNA rearrangements.
   Claudia Martini, Beatrice Chelli, Laura Betti, Marina Montali, Michelangelo Mancuso, Gino Giannaccini
   Molecular medicine (Cambridge, Mass.) 2002 IF: 8.30 Q1 PMID: 12606819

   The peripheral-type benzodiazepine receptors (PBR) are localized on the outer mitochondrial membrane, as a constituent of mitochondrial permeability transition (MPT)-pore. Among its hypothesized funct…

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8. Angiotensin II-induced mesangial cell apoptosis: role of oxidative stress.
   Saurabh Lodha, Dhimant Dani, Rajeev Mehta, Madhu Bhaskaran, Krishna Reddy, Guohua Ding
   Molecular medicine (Cambridge, Mass.) 2002 IF: 8.30 Q1 PMID: 12606818

   Angiotensin II (ANG II) has been shown to play a role in the induction of glomerular injury. In the present study, we evaluated the effects of ANG II on mesangial cell apoptosis and the involved molec…

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9. Semicarbazide-sensitive amine oxidase (SSAO) gene expression in alloxan-induced diabetes in mice.
   Jenny E L Nordquist, Camilla Göktürk, Lars Oreland
   Molecular medicine (Cambridge, Mass.) 2002 IF: 8.30 Q1 PMID: 12606817

   Plasma activity of semicarbazide-sensitive amine oxidase (SSAO) has been reported to be significantly higher in diabetic patients compared to healthy controls. Due to the production of highly angiotox…

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10. Visualization of irreparable ischemic damage in brain by selective labeling of double strand blunt-ended DNA breaks.
    Vladimir V Didenko, Hop Ngo, Candace L Minchew, Denise J Boudreaux, Marsha A Widmayer, David S Baskin
    Molecular medicine (Cambridge, Mass.) 2002 IF: 8.30 Q1 PMID: 12606816

    Double-strand DNA breaks with blunt ends represent the most serious type of DNA damage, and cannot be efficiently repaired by cells. They are generated in apoptosis or necrosis and are absent in norma…

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