SLEEP睡眠
SLEEP(英文缩写 SLEEP),ISSN 0161-8105,eISSN 1550-9109,中文译名:睡眠 是一本学术期刊。本页汇总该期刊的最新影响因子、分区信息以及最新收录于 PubMed 的文献,帮助您快速了解期刊全貌。
发文量统计区间：2025-09-28 至 2026-09-28，按本站收录文献的发表日期统计。
期刊介绍
历年影响因子趋势
| JCR 数据年份 | 影响因子 | JCR 分区 |
|---|---|---|
| 2021 | 6.313 | Q1 |
| 2022 | 5.600 | Q1 |
| 2023 | 5.300 | Q1 |
| 2024 | 4.900 | Q1 |
| 2025 | 7.000 | Q1 |
SLEEP 最新收录文献
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1. Distinct Thalamostriatal Functional Network Dysregulation in Comorbid Insomnia and Sleep Apnea.
PMID:日期:2026-09-25Comorbid insomnia and sleep apnea (COMISA) is a prevalent and clinically challenging phenotype, yet the neural substrates distinguishing it from isolated obstructive sleep apnea (OSA) remain elusive. We employed a multimodal graph theoretical approach to identify the structural and functional network patterns associated with the COMISA phenotype. We analyzed 124 male participants (45 controls, 58 OSA, and 21 COMISA). Structural covariance networks were derived from regional gray matter volume data, and functional networks from resting-state fMRI. Graph metrics-including strength, clustering coefficient (CC), efficiency, and closeness centrality (CloC)-were quantified for weighted, undirected networks to assess global and nodal topological properties. Functional network analysis revealed significant global hyper-connectivity in both patient groups compared to controls. Crucially, COMISA demonstrated a distinct functional pattern compared to OSA, characterized by significantly elevated nodal metrics (CC and CloC) within thalamostriatal circuits, specifically involving the bilateral thalamus and putamen. Exploratory analyses across the cohort suggested possible positive associations between these nodal metrics and the severity of insomnia and depressive symptoms. In contrast, structural covariance networks showed no significant group differences, indicating a dissociation between the two modalities. Our findings suggest that COMISA presents a distinct functional pattern, which is consistent with circuit-level dysregulation, specifically within the thalamostriatal axis. This subcortical pattern within arousal-modulating circuits offers a potential pathophysiological explanation for the clinical complexity and treatment resistance frequently observed in COMISA.
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2. Insomnia with objective short sleep duration: A biomarker for mortality risk, not yet a mandate for practice.
PMID:日期:2026-09-25该文献暂无摘要。
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3. Comorbid insomnia and sleep apnea: A potential risk factor for neurodegenerative disease?
PMID:日期:2026-09-24该文献暂无摘要。
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4. Sleep regularity and efficiency are associated with nocturnal glycemic control in pregnant women with gestational diabetes.
PMID:日期:2026-09-24Nocturnal glycemic control is a critical component of blood glucose management for pregnant women with gestational diabetes mellitus (GDM). This study aims to investigate the associations of sleep quality with nocturnal glycemic control among GDM participants. This study included 1,186 GDM participants with available data from the WeBirth cohort in Hangzhou, China. Sleep quality and blood glucose levels were assessed simultaneously over 14 days using accelerometers and continuous glucose monitoring (CGM) devices. Multivariate linear mixed-effects models were applied to examine the associations of the sleep regularity index (SRI) and sleep efficiency (SE) with CGM-derived nocturnal metrics, adjusting for potential confounders (e.g., maternal age, pre-pregnancy BMI and gestational weeks). Per 1-SD increment in SRI was associated with lower nocturnal glycemic metrics, including coefficient of variation (β = -0.11 SD; 95% CI, -0.13 to -0.08) and time-above-range (β = -0.08 SD; 95% CI, -0.10 to -0.05). SE demonstrated comparable associations with these metrics (coefficient of variation: β = -0.13 SD; 95% CI, -0.16 to -0.11; time-above-range: β = -0.10 SD; 95% CI, -0.12 to -0.08). Further joint analyses indicated that SE exerts an independent, additive effect beyond sleep regularity. Specifically, individuals characterized by both irregular and inefficient sleep exhibited substantially higher mean blood glucose levels compared to those with regular and efficient sleep (β = 0.34 SD; 95% CI, 0.21 to 0.47). This study highlights that regular and efficient sleep patterns are independently and jointly associated with more favourable nocturnal glycemic outcomes in pregnant women with GDM.
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5. Sleep Apnea and Risk of Comorbid Insomnia in Arab, Black, and White Women and Men; A Longitudinal Study.
PMID:日期:2026-09-24Comorbid insomnia and sleep apnea (COMISA) results in worse health outcomes than either condition alone. While the bidirectional link between obstructive sleep apnea (OSA) and insomnia is established, the causal trajectory from OSA to incident insomnia - and potential disparities by sex, race, and ethnicity - remains understudied. We investigated the association between OSA and incident insomnia within a large, diverse clinical cohort. We conducted a retrospective cohort study of 313,953 adults (aged 40-85 years) within the Electronic Medical Records (EMR) of the University of Michigan Health System (2012-2023). Participants were required to be insomnia-free for one year following cohort entry. Arab ethnicity was identified using a validated surname-based algorithm, while Black and White designations were based on EMR self-report.To estimate the risk of incident insomnia following an OSA diagnosis, we employed multivariable Cox proportional hazards models adjusted for age, sex, race and ethnicity, BMI, and Area Deprivation Index, a measure of socioeconomic disadvantage. Interaction terms were included to evaluate whether the association between OSA and insomnia differed significantly across sex, racial, and ethnic subgroups. OSA diagnosis was associated with a significantly increased risk of subsequent insomnia. Although these associations did not vary by sex, significant interactions were observed according to race and ethnicity (interaction term p<0.04). Arab (HR=1.57; 95% CI [1.34-1.84]) and Black patients (HR=1.57; 95% CI [1.45-1.70]) exhibited a higher hazard of incident insomnia than White patients (HR=1.39; 95% CI [1.35-1.43]). These findings demonstrate that Arab and Black patients face a disproportionately higher risk of developing COMISA following OSA diagnosis.
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6. Effects of light exposure on psychomotor vigilance in shift workers: a systematic review.
PMID:日期:2026-09-23Circadian misalignment caused by shift work has been linked to fatigue and diminished psychomotor performance. Light, the major zeitgeber of circadian rhythms, has been investigated in studies, particularly regarding the spectral composition's effect on vigilance. This review explores whether light interventions improve reaction times and reduce errors such as false starts and lapses among shift workers. Following PRISMA guidelines, a systematic search was conducted in PubMed, PsycINFO, Scopus, and Web of Science. The Cochrane Risk of Bias 2 tool and ROBINS-I tool were used to assess the risk of bias. We included peer-reviewed studies that provided descriptions of lighting conditions, including illuminance (in lx), color temperature (in K), and melanopic equivalent daylight illuminance (in MEDI lx). The primary outcome was mean reaction time, with secondary measures including lapses, false starts, and 10% fastest and slowest reaction times. Differences in light parameters (lx, K, and MEDI lx) between the intervention and control groups were summarized narratively. Ratios of intervention to control values were calculated, adjusting for exposure duration. The search resulted in 3,120 records, of which 25 studies met inclusion criteria. Ten studies reported significant association between reaction times and lighting. Light exposures of at least 5 hours, combined with a minimum difference of 159 MEDI lx, were associated with a statistically significant improvement in reaction times. Extended light exposure combined with a higher melanopic gradient appears to enhance psychomotor vigilance. However, heterogeneity in study design and reporting of intervention studies limits the certainty of this conclusion.
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7. The Neurobiological Construct(s) of COMISA: A Network Perspective on Phenotype(s) and Mechanism(s).
PMID:日期:2026-09-19该文献暂无摘要。
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8. "The Better I Sleep, The Better My Memory": Sleep-Health Education Through Experiential Learning.
PMID:日期:2026-09-18该文献暂无摘要。
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10. Multimodal Respiratory Event Detection Leveraging Signal Complementarity under Intermittent SpO₂ Loss.
PMID:日期:2026-09-18Current multimodal approaches for automated sleep apnea detection usually assume continuous signal availability, although sensor dropout is common in clinical practice. We evaluated probability-level late fusion across signal configurations and missing-data conditions to determine whether performance depends more on signal complementarity or modality count. We trained random forest base classifiers on EEG, ECG, SpO₂, and abdominal effort features from 148 patients. Each classifier generated three-class posterior probabilities, which were concatenated and input to a meta-classifier for final epoch-level prediction. Unreliable SpO₂ epochs were excluded only from SpO₂ base-model fitting but retained during meta-classifier training and testing using uniform placeholder probabilities [0.333, 0.333, 0.333] plus the isBad quality flag. Selected unimodal and multimodal configurations were evaluated in 37 held-out patients using macro-F1 as the primary metric. Bootstrap confidence intervals stratified by SpO₂ quality assessed robustness under dropout. Abdominal effort alone achieved macro-F1 = 0.996. SpO₂ + abdominal effort achieved the highest macro-F1 = 0.997, only marginally above abdominal effort alone. ECG + EEG outperformed SpO₂ + ECG and SpO₂ + EEG despite lacking direct respiratory or oxygenation information. SpO₂ + EEG showed degraded performance (macro-F1 = 0.739; hypopnea precision = 0.203). Adding SpO₂ to ECG + EEG reduced performance (0.872 vs 0.899). Performance remained stable despite 30.7% SpO₂ dropout. Within probability-level late fusion, performance depended more on signal complementarity than modality count. Quality-aware probability integration enabled robust classification under realistic SpO₂ dropout without retraining, but fixed-epoch late fusion limited exploitation of temporally misaligned oximetry information.