eFood电子食品

eFood(英文缩写 EFOOD),ISSN 2666-3066,eISSN 2666-3066,中文译名:电子食品 是一本学术期刊。本页汇总该期刊的最新影响因子、分区信息以及最新收录于 PubMed 的文献,帮助您快速了解期刊全貌。

2026 年数据 · 影响因子
5.700
JCR 分区
Q1
CAS 分区
B3
近一年发文量
0
本站 PubMed 收录统计

发文量统计区间:2025-10-01 至 2026-09-30,按本站收录文献的发表日期统计。

ISSN: 2666-3066 · eISSN: 2666-3066 · 缩写: EFOOD ·中文: 电子食品

期刊介绍

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期刊简介

eFood 是一本聚焦食品科学与技术前沿的国际英文期刊,关注食品化学、营养、安全及加工技术等交叉领域。其读者群包括食品科研人员、营养学者、产业研发人员及政策制定者,旨在促进从基础研究到应用转化的学术交流。

研究方向

主要方向涵盖食品成分与功能、食品安全与检测、食品加工与保藏、营养与健康、食品生物技术及可持续食品体系。论文类型以原创研究为主,兼有综述、短通讯和方法学文章,鼓励跨学科与新技术应用研究。

期刊特色

研究取向偏重创新性与应用潜力,强调实验数据扎实和机制探讨。论文通常要求明确的研究问题和较完整的验证。适合食品科学、营养学、生物技术等领域的研究生、教师及企业研发人员阅读与投稿。

投稿难度

投稿难度中等偏上,对创新性和数据完整性要求较高。建议在实验设计、统计分析和英文表达上充分打磨,并突出与现有文献的差异。不能仅凭分区判断录用难易,需结合具体研究方向与期刊近期发表内容评估。

eFood 最新收录文献

  1. JCR分区: Q1 CAS分区: B3 影响因子: 5.7

    1. Effects of Dietary Interventions on Gut Microbiota in Humans and the Possible Impacts of Foods on Patients' Responses to Cancer Immunotherapy.

    作者:
    Li-Shu Wang, Yue Yang Mo, Yi-Wen Huang, Carla Elena Echeveste, Hsin-Tzu Wang, Jiali Chen, Kiyoko Oshima, Martha Yearsley, Jesus Simal-Gandaraf, Maurizio Battino, Jianbo Xiao, Jiebiao Chen, Chongde Sun, Jianhua Yu, Weibin Bai
    期刊:
    日期:
    2020-08-27

    The gut microbiota-the community of microorganisms in the gut-has been implicated in many physical and mental disorders in addition to intestinal diseases. Diets are the most studied and promising factors for altering it. Indeed, certain dietary interventions that increase fiber intake rapidly change levels of certain nutrients that can modify the composition of the microbiota, promoting richness and diversity. Recent intriguing evidence from several human clinical trials suggested that the composition and diversity of patients' gut microbiotas at baseline can influence their responses to cancer immunotherapy. If the factors that influence the gut microbiota were fully understood, it is conceivable that manipulating them could boost therapeutic responses in cancer patients. In this review, we investigate the possibility of using fruits, vegetables, or whole grains to enhance response to cancer therapies in humans, as current evidence suggests that these dietary components can manipulate and enhance diversity of the gut microbiota. Accordingly, dietary interventions with locally available fruits, vegetables, and whole grains might be an affordable and safe approach to enhancing the diversity of the gut microbiota before immunotherapy, in turn improving patients' responses to their treatments.

  2. JCR分区: Q1 CAS分区: B3 影响因子: 5.7

    2. {"_":"Can Natural Products Suppress Resistant to Fight Against Gastric Diseases in Humans?","i":["Helicobacter pylori"]}

    作者:
    Li-Shu Wang, Carla Elena Echeveste, Jianhua Yu, Yi-Wen Huang, John Lechner, Ling Mei, Patrick Sanvanson, Martha Yearsley, Chin-Kun Wang, Gary Stoner
    期刊:
    日期:
    2020-02-19

    More than 50% of the world's population is infected with Helicobacter pylori. is the major causative agent of gastric ulcers and gastric cancer. eradication using antibiotics either alone or together with a proton pump inhibitor is the primary strategy to decrease the incidence of gastric cancer. Although eradication therapy is effective, there are significant adverse effects and more importantly, resistance to antibiotics occurs, which represents a major therapeutic challenge. Multiple natural products have been shown to suppress both and in animal model systems. However, only a handful of natural products have been evaluated in human clinical trials. The focus of this review is to summarize the results of published human clinical trials to assess the ability of natural products to reduce or eliminate infections. Current evidence suggests that these products appear to have great potential to be developed as pharmaceutical candidates for eradication of , hopefully both antibiotic-sensitive and antibiotic-resistant strains. Frequent consumption of locally produced foodstuff for controlling infection in different countries around the world may well be a feasible long-term solution to fight against this worldwide prevalent pathogen.

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