Nature Reviews Clinical Oncology

Nature Reviews Clinical Oncology(英文缩写 NAT REV CLIN ONCOL),ISSN 1759-4774,eISSN 1759-4782 是一本学术期刊。本页汇总该期刊的最新影响因子、分区信息以及最新收录于 PubMed 的文献,帮助您快速了解期刊全貌。

2026 年数据 · 影响因子
94.600
JCR 分区
Q1
CAS 分区
B1
近一年发文量
0

指标来源:jcr_cas_ifqb

ISSN: 1759-4774 · eISSN: 1759-4782 · 缩写: NAT REV CLIN ONCOL

期刊简介

暂无简介。

历年影响因子趋势

年份影响因子JCR 分区
-Q1
-Q1
-Q1
-Q1
-Q1

Nature Reviews Clinical Oncology 最新收录文献

※ 中文译文由 AI 辅助生成,仅供学术参考,请以英文原文为准。

  1. HCT-ASCT confirmed as standard-of-care consolidation therapy for PCNSL.
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  2. Benefit from perioperative enfortumab vedotin-pembrolizumab in cisplatin-eligible MIBC.
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  3. More options, more questions in the management of early relapsed and/or refractory multiple myeloma.
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  4. Clinical toxicity of ADCs and ICI-ADC combinations: mechanisms, patterns and management.

    Antibody-drug conjugates (ADCs) and immune checkpoint inhibitors (ICIs) have reshaped cancer therapy both as monotherapy and in combination. However, these drug classes have toxicity profiles that, de…

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  5. De-escalation trials in multiple myeloma: past, present and future. 多发性骨髓瘤降级试验:过去、现在和未来

    De-escalation of therapy constitutes a new phase in the development of therapeutic approaches for patients with multiple myeloma (MM) and is aimed at maintaining durable disease control while reducing…

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  6. Electronic cigarettes, smoking and lung cancer risk: some evidence about the continuum of harm.
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  7. Towards liquid biopsy-based analysis of antitumour immunity.

    Analysis of tissue biopsy samples is the gold-standard approach to cancer diagnosis and patient selection for biomarker-guided therapies. Although spatial analyses of tumour tissue can provide importa…

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  8. Does OptiTROP-Lung05 open a new frontier for first-line ADC-ICI combinations in NSCLC?
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  9. {"_":"Second-line Lu-edotreotide shows promise in GEP NETs.","sup":["177"]}
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  10. CRISPR in clinical oncology: translational advances from molecular diagnostics to therapeutics.

    Cancer care is increasingly driven by molecular classification, yet many key oncogenic drivers remain undruggable, and intrinsic or acquired resistance to treatment frequently limits durable clinical …

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