Journal of Neuroinflammation
Journal of Neuroinflammation(英文缩写 J NEUROINFLAMM),ISSN 1742-2094,eISSN 1742-2094 是一本学术期刊。本页汇总该期刊的最新影响因子、分区信息以及最新收录于 PubMed 的文献,帮助您快速了解期刊全貌。
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期刊简介
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Journal of Neuroinflammation 最新收录文献
※ 中文译文由 AI 辅助生成,仅供学术参考,请以英文原文为准。
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Estrogen deprivation exacerbates Alzheimer's disease pathology through neuronal CTSS signaling.
Alzheimer's disease (AD) exhibits a pronounced sex bias, with women facing disproportionately higher risk and more severe pathology. Postmenopausal estrogen decline is implicated in this vulnerability…
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{"_":"The kallikrein-kinin system modulates CD4 T cell function and contributes to T cell migration into the brain after stroke.","sup":["+"]}
Ischemic stroke elicits a sustained thromboinflammatory response that critically shapes secondary brain injury. The kallikrein-kinin system (KKS) has been identified as the interface of vascular injur…
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Editorial Expression of Concern: Biliverdin reductase-A attenuated GMH induced inflammatory response in the spleen by inhibiting toll-like receptor-4 through eNOS/NO pathway.
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The locus coeruleus at the crossroads of inflammation and neurodegeneration in multiple sclerosis.
Multiple sclerosis (MS) is increasingly recognized as a systems-level disorder encompassing grey matter injury, network disconnection, and neuropsychiatric symptoms alongside demyelination. Within thi…
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Retraction Note: Neuroinflammation is associated with infiltration of T cells in Lewy body disease and α-synuclein transgenic models.
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Correction: Adenosine A3 receptor as a novel therapeutic target to reduce secondary events and improve neurocognitive functions following traumatic brain injury.
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NLRC5 restricts Japanese encephalitis virus replication and neuroinflammation by interacting with the viral NS3 protein in an IFN-γ-dependent manner. NLRC5通过以IFN-γ依赖的方式与病毒NS3蛋白相互作用来限制乙型脑炎病毒的复制和神经炎症
NLRC5 is a known activator of MHC class I genes and a regulator of type I interferon activity. Although its expression increases in brains infected with Japanese encephalitis virus (JEV), its specific…
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CX3CL1/CX3CR1-dependent microglial phagocytosis of oligodendrocyte precursor cells contributes to sevoflurane-induced myelination impairments in neonatal mice.
General anesthesia exposure in early life may disrupt the normal progression of developmental myelination, but the underlying mechanisms remain unclear. Early postnatal microglia in developing white m…
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CXCL10 contributes to female-specific pathological progression in tauopathy model mice.
Neuroinflammation plays a central role in the progression of tauopathy via the glial activation and T cell accumulation in the brain parenchyma. However, the key molecular mediators that link these pr…
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Neuroinflammation-associated thalamocortical functional connectivity and cognition in older adults. 老年人神经炎症相关的丘脑皮层功能连接与认知
Neuroinflammation has detrimental neurocognitive effects in patients with systemic and neuropsychiatric diseases. Yet, outside of these conditions, it is unclear to what extent neuroinflammation modul…