SEED SCIENCE RESEARCH种子科学研究

SEED SCIENCE RESEARCH(英文缩写 SEED SCI RES),ISSN 0960-2585,eISSN 1475-2735,中文译名:种子科学研究 是一本学术期刊。本页汇总该期刊的最新影响因子、分区信息以及最新收录于 PubMed 的文献,帮助您快速了解期刊全貌。

2026 年数据 · 影响因子
2.100
JCR 分区
Q2
CAS 分区
B3
近一年发文量
0
本站 PubMed 收录统计

发文量统计区间:2025-10-01 至 2026-09-30,按本站收录文献的发表日期统计。

ISSN: 0960-2585 · eISSN: 1475-2735 · 缩写: SEED SCI RES ·中文: 种子科学研究

期刊介绍

选择期刊介绍栏目

期刊简介

《Seed Science Research》是种子生物学领域的国际同行评议期刊,聚焦种子发育、休眠与萌发、活力与寿命、贮藏与劣变、种子生态及进化等基础与应用问题。读者群包括植物科学家、农学与园艺研究者、种质资源保护人员及种子产业技术人员,强调机制性发现与实验证据,为种子科学提供跨学科交流平台。

研究方向

主要发表种子发育与成熟、休眠与萌发调控、活力与耐逆性、贮藏寿命与劣变机制、种子生态与进化、种质保存及萌发技术等方向的研究。论文类型以原创研究为主,兼有综述、短通讯和方法学文章,鼓励分子、生理与生态层面的整合研究。

期刊特色

研究取向偏重机制解析与实验验证,要求数据扎实、结论明确,对种子生物学理论或应用有实质推进。论文通常结合生理、生化、分子或生态手段,适合从事种子科学、作物学、植物生理生态及种质资源研究的科研人员与研究生阅读参考。

投稿难度

投稿难度中等偏上,期刊重视机制深度与实验严谨性,而非仅凭分区判断。建议在投稿前明确科学问题、完善对照与统计、充分讨论与既有文献的差异,并针对种子生物学读者清晰阐述创新点,语言与图表规范也需仔细打磨。

历年影响因子趋势

JCR 数据年份影响因子JCR 分区
20211.585Q3
20222.100Q2
20232.100Q2
20241.900Q2
20252.100Q2

SEED SCIENCE RESEARCH 最新收录文献

  1. JCR分区: Q2 CAS分区: B3 影响因子: 2.1

    1. Functional analysis of TaABF1 during abscisic acid and gibberellin signalling in aleurone cells of cereal grains.

    作者:
    Lauren J Harris, Sarah A Martinez, Benjamin R Keyser, William E Dyer, Russell R Johnson
    日期:
    2013-06

    The wheat transcription factor TaABF1 physically interacts with the protein kinase PKABA1 and mediates both abscisic acid (ABA)-induced and ABA-suppressed gene expression. In bombarded aleurone cells of imbibing grains, the effect of TaABF1 in down-regulating the gibberellin (GA)-induced promoter was stronger in the presence of exogenous ABA. As these grains contained low levels of endogenous ABA, the effect of TaABF1 may also be mediated by ABA-induced activation even in the absence of exogenous ABA. Levels of TaABF1 protein decreased slightly during imbibition of afterripened grains. However, TaABF1 levels (especially in aleurone layers) were not substantially affected by exogenous ABA or GA, indicating that changes in TaABF1 protein level are not an important part of regulating its role in hormone signalling. We found that TaABF1 was phosphorylated in aleurone cells, suggesting a role for post-translational modification in regulating TaABF1 activity. Induction of by overexpression of the transcription factor GAMyb could not be prevented by TaABF1, indicating that TaABF1 acts upstream of transcription in the signalling pathway. Supporting this view, knockdown of TaABF1 by RNA interference resulted in increased expression from the promoter. These results are consistent with a model in which TaABF1 is constitutively present in aleurone cells, while its ability to down-regulate is regulated in response to ABA.

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