BASIC RESEARCH IN CARDIOLOGY

BASIC RESEARCH IN CARDIOLOGY(英文缩写 BASIC RES CARDIOL),ISSN 0300-8428,eISSN 1435-1803 是一本学术期刊。本页汇总该期刊的最新影响因子、分区信息以及最新收录于 PubMed 的文献,帮助您快速了解期刊全貌。

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BASIC RESEARCH IN CARDIOLOGY 最新收录文献

1. Transforming growth factor beta inhibition increases mortality and left ventricular dilatation after myocardial infarction.
   Stefan Frantz, Kai Hu, Anna Adamek, Jürgen Wolf, Abed Sallam, Sebastian K G Maier
   Basic research in cardiology 2008 IF: 9.20 Q1 PMID: 18651091

   Transforming growth factor (TGF)-beta is a locally generated cytokine involved in healing processes and tissue fibrosis, all relevant for cardiac remodeling and the development of heart failure after …

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2. Acidosis, oxygen, and interference with mitochondrial permeability transition pore formation in the early minutes of reperfusion are critical to postconditioning's success.
   Michael V Cohen, Xi-Ming Yang, James M Downey
   Basic research in cardiology 2008 IF: 9.20 Q1 PMID: 18626679

   Repetitive cycles of reflow/reocclusion in the initial 2 min following release of a prolonged coronary occlusion, i.e., ischemic postconditioning (IPoC), salvages ischemic myocardium. We have proposed…

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3. Maturation-induces endothelial dysfunction via vascular inflammation in diabetic mice.
   Cuihua Zhang, Yoonjung Park, Andrea Picchi, Barry J Potter
   Basic research in cardiology 2008 IF: 9.20 Q1 PMID: 18600366

   We hypothesized that maturation-induced vascular inflammation produces endothelial dysfunction in type II diabetes and TNFalpha plays a key role in triggering inflammation in the development of diabet…

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4. Persistent beneficial effect of postconditioning against infarct size: role of mitochondrial K(ATP) channels during reperfusion.
   James Mykytenko, James G Reeves, Hajime Kin, Ning-Ping Wang, Amanda J Zatta, Rong Jiang
   Basic research in cardiology 2008 IF: 9.20 Q1 PMID: 18600365

   This study tested the hypothesis that inhibition of myocardial injury and modulation of mitochondrial dysfunction by postconditioning (Postcon) after 24 h of reperfusion is associated with activation …

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5. The role of inflammatory cytokines in endothelial dysfunction.
   Cuihua Zhang
   Basic research in cardiology 2008 IF: 9.20 Q1 PMID: 18600364

   Clinical and experimental data support a link between endothelial dysfunction and inflammation. Inflammatory cytokines are important protagonists in formation of atherosclerotic plaque, eliciting effe…

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6. Clinical translation of cardioprotective strategies : report and recommendations of the Hatter Institute 5th International Workshop on Cardioprotection.
   Derek J Hausenloy, Derek M Yellon
   Basic research in cardiology 2008 IF: 9.20 Q1 PMID: 18600363
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7. Inhibiting matrix metalloproteinase-2 reduces protein release into coronary effluent from isolated rat hearts during ischemia-reperfusion.
   Justyna Fert-Bober, Hernando Leon, Jolanta Sawicka, Rashpal S Basran, Richard M Devon, Richard Schulz
   Basic research in cardiology 2008 IF: 9.20 Q1 PMID: 18512095

   Previous studies have shown that the disruption of the coronary endothelium and the increase in its permeability during ischemia-reperfusion (I/R), are linked to matrix metalloproteinase-2 (MMP-2) act…

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8. Activation of kappa-opioid receptors at reperfusion affords cardioprotection in both rat and mouse hearts.
   Jason N Peart, Eric R Gross, Melissa E Reichelt, Anna Hsu, John P Headrick, Garrett J Gross
   Basic research in cardiology 2008 IF: 9.20 Q1 PMID: 18500486

   The temporal properties of kappa-opioid receptor (kappa-OR) mediated cardioprotection are less well characterised than delta-opioid receptor (delta-OR) responses. This study was aimed at delineating t…

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9. Signal transducer and activator of transcription 3 is involved in the cardioprotective signalling pathway activated by insulin therapy at reperfusion.
   Britt N Fuglesteg, Naushaad Suleman, Crina Tiron, Tambuzai Kanhema, Lydia Lacerda, Thomas V Andreasen
   Basic research in cardiology 2008 IF: 9.20 Q1 PMID: 18500485

   To evaluate the significance of the JAK-STAT pathway in insulin-induced cardioprotection from reperfusion injury. In isolated perfused rat hearts subjected to insulin therapy (0.3 mU/ml) +/- AG490 (5 …

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10. NO and PGI(2) in coronary endothelial dysfunction in transgenic mice with dilated cardiomyopathy.
    Lukasz Drelicharz, Valery Kozlovski, Tomasz Skorka, Sylwia Heinze-Paluchowska, Andrzej Jasinski, Anna Gebska
    Basic research in cardiology 2008 IF: 9.20 Q1 PMID: 18431525

    The aim of the present work was to analyze coronary endothelial function in the transgenic mouse model of dilated cardiomyopathy (Tgalphaq*44 mice). Coronary vasodilatation, both NO-dependent (induced…

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