BLOOD
BLOOD(英文缩写 BLOOD),ISSN 0006-4971,eISSN 1528-0020 是一本学术期刊。本页汇总该期刊的最新影响因子、分区信息以及最新收录于 PubMed 的文献,帮助您快速了解期刊全貌。
指标来源:jcr_cas_ifqb
期刊简介
暂无简介。
历年影响因子趋势
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BLOOD 最新收录文献
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Abstracts of the American Society of Hematology 53rd Annual Meeting, December 10-13, 2011, San Diego, California, USA.
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Abstracts of the American Society of Hematology 51st Annual Meeting, December 5-8, 2009, New Orleans, Louisiana, USA.
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Abstracts of the American Society of Hematology 50th Annual Meeting, December 6-9, 2008, San Francisco, California, USA.
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AIDS, T cells, chemotherapy: HAART-breaking?
Chemotherapy depletes T cells in AIDS patients on HAART, but preexisting lymphopenia limits the scale compared with non-AIDS patients. Thus T-cell recovery time to baseline may be rapid, but the risk …
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Cell expansion and maintenance of stemness.
Manipulation of hematopoietic cells to expand output while maintaining stem-cell potential has been an elusive goal of experimental hematology. The development of a system using a chemically induced d…
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VWD type 1: a calculated diagnosis.
Tosetto and colleagues have developed a mathematical method to quantify the odds of having type 1 von Willebrand Disease based on a person's family history, bleeding score, and VWF level.
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Gardos pathway to sickle cell therapies?
In this issue of Blood, Ataga and colleagues report that treatment of sickle cell disease patients with senicapoc, a Gardos channel inhibitor, reduces the number of dehydrated cells, increases hemoglo…
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Osteopoietic stem cells: transplantable, but regeneratively limited.
Questions about the transplantability of mesenchymal stem cells, their ability to engraft within the bone marrow of recipients, and hence their clinical usefulness have been hotly debated for several …
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AML1 and Evi1: coconspirators in MDS/AML?
In this issue of Blood, Watanabe-Okochi and colleagues use a mouse bone marrow transplantation model to demonstrate that mutant alleles of AML1 (RUNX1) can initiate a myelodysplastic syndrome (MDS) th…
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Below the NK-cell surface: PIP2.
In this issue Blood, Micucci and colleagues demonstrate that PIP2 plays an essential role in NK-cell cytotoxicity by controlling the release of lytic mediators at the NK cell-target interface.