ARCHIVES OF DERMATOLOGICAL RESEARCH皮肤病学研究档案

ARCHIVES OF DERMATOLOGICAL RESEARCH(英文缩写 ARCH DERMATOL RES),ISSN 0340-3696,eISSN 1432-069X,中文译名:皮肤病学研究档案 是一本学术期刊。本页汇总该期刊的最新影响因子、分区信息以及最新收录于 PubMed 的文献,帮助您快速了解期刊全貌。

2026 年数据 · 影响因子
2.500
JCR 分区
Q2
CAS 分区
B4
近一年发文量
5
本站 PubMed 收录统计

发文量统计区间:2025-09-28 至 2026-09-28,按本站收录文献的发表日期统计。

ISSN: 0340-3696 · eISSN: 1432-069X · 缩写: ARCH DERMATOL RES ·中文: 皮肤病学研究档案

期刊介绍

选择期刊介绍栏目

期刊简介

《Archives of Dermatological Research》是一本历史悠久的国际皮肤病学学术期刊,发表皮肤基础与临床研究。内容涵盖皮肤生物学、免疫学、病理学、遗传学及治疗学,关注炎症性皮肤病、皮肤肿瘤和感染等方向。读者群主要为皮肤科医师、科研人员及相关专业研究生,适合希望了解皮肤病机制与临床转化进展的读者。

研究方向

主要刊载皮肤病学领域的原创研究论文,包括实验性皮肤生物学、皮肤免疫与炎症、皮肤肿瘤发生机制、遗传性皮肤病、伤口愈合及新型治疗手段等主题。也接受简要报告和综述类稿件,强调研究设计的科学性与结果的临床相关性。

期刊特色

研究取向兼顾基础实验与临床观察,论文通常要求有明确机制探讨或临床意义,数据完整、方法透明。适合皮肤科临床医生、基础医学研究者及从事皮肤相关药物开发的人员阅读与投稿,对跨学科转化研究较为友好。

投稿难度

投稿难度中等偏上,期刊对研究新颖性和方法学质量有一定要求,但并非仅凭分区判断。建议准备充分的前期数据、清晰的机制阐述和规范的统计学分析,并在语言和格式上认真打磨,以提升送审与录用机会。

历年影响因子趋势

JCR 数据年份影响因子JCR 分区
20213.033Q2
20223.000Q2
20231.800Q3
20242.100Q3
20252.500Q2

ARCHIVES OF DERMATOLOGICAL RESEARCH 最新收录文献

  1. JCR分区: Q2 CAS分区: B4 影响因子: 2.5
  2. JCR分区: Q2 CAS分区: B4 影响因子: 2.5
  3. JCR分区: Q2 CAS分区: B4 影响因子: 2.5
  4. JCR分区: Q2 CAS分区: B4 影响因子: 2.5
  5. JCR分区: Q2 CAS分区: B4 影响因子: 2.5

    5. Increased Risk of Melanoma and Basal Cell Carcinoma in Patients with Sjogren's Syndrome: A Nested Case-Control Study.

    作者:
    Vineeth R Vaidyula, Raphaella Lambert, Omar Alani, Benjamin Ungar, Nicholas Gulati, Jonas A Adalsteinsson
    日期:
    2025-12-01

    该文献暂无摘要。

  6. JCR分区: Q2 CAS分区: B4 影响因子: 2.5

    6. Mohs micrographic surgery is non-inferior to wide local excision for disease specific survival in sebaceous carcinoma: analysis of the Surveillance, Epidemiology, and End Results (SEER) database (2000-2021).

    6. 在皮脂腺癌的疾病特异性生存率方面,莫氏显微手术不亚于广泛的局部切除:监测、流行病学和最终结果(SEER)数据库分析(2000-2021)
    作者:
    Akshay Pulavarty, Lynn Liu, Michelle Juarez, Maressa C Criscito, Nayoung Lee, Mary Stevenson, John Carucci
    日期:
    2025-06-11

    该文献暂无摘要。

  7. JCR分区: Q2 CAS分区: B4 影响因子: 2.5

    7. Nicotinamide for secondary keratinocyte carcinoma prevention in solid organ transplant recipients.

    作者:
    Jonathan C Hwang, Kevin T Savage, Melissa Pugliano-Mauro
    日期:
    2025-06-11

    Nicotinamide has many well-established chemopreventive properties in protecting against ultraviolet-induced skin damage, mitigating inflammation, and reducing keratinocyte carcinoma (KC) development among immunocompetent individuals. Its effectiveness in immunosuppressed patients, however, is unclear. There is conflicting research on whether nicotinamide effectively decreases KCs in immunosuppressed solid organ transplant recipients (SOTRs). This study assesses the effectiveness of nicotinamide in the secondary prevention of KC in immunosuppressed patients. We conducted a retrospective cohort study in a single tertiary care institution. The primary outcome was KC incidence in the year before and after nicotinamide supplementation. Secondary outcomes included the incidence of invasive squamous cell carcinoma (SCC), SCC in situ (SCCis), and basal cell carcinoma (BCC) over one- and two-year intervals. All included patients had taken oral nicotinamide, 500 milligrams twice daily, for at least one year. A total of 47 SOTRs (74.5% male; mean age 65.2 years) were included in our retrospective cohort study. Of 81 patients initially screened, 34 were excluded due to inadequate follow-up, dermatologic care outside our institution, or early discontinuation of nicotinamide. At one year post-nicotinamide supplementation, total KC incidence decreased from 224 (78 SCC, 103 SCCis, 43 BCC) to 121 cases (40 SCC, 55 SCCis, 26 BCC), a mean reduction of 2.19 KCs (95% CI: -3.48 to -0.90; p = 0.0012). Significant reductions were observed in SCC (mean decrease of 1.15; 95% CI: -1.78 to -0.52; p = 0.00081) and SCCis (mean decrease of 1.37; 95% CI: -2.61 to -0.13; p = 0.032). BCC reduction was not statistically significant (p = 0.13). In the 31 patients with two-year follow-up data, KC incidence declined from 234 to 167, a mean reduction of 2.18 KCs (95% CI: -4.18 to -0.14; p = 0.037). Sensitivity analyses excluding patients on concomitant acitretin confirmed that reductions in total KC incidence maintained significance at both one-year and two-year intervals. Nicotinamide supplementation significantly decreased KC incidence in immunosuppressed SOTRs over the one-year and two-year intervals. We recommend nicotinamide as a low-risk, low-cost chemopreventive supplement for reducing KCs in SOTRs.

  8. JCR分区: Q2 CAS分区: B4 影响因子: 2.5

    8. Off-label use of biologics and janus kinase (JAK) inhibitors for scarring alopecias: a narrative review.

    8. 标签外使用生物制剂和janus激酶(JAK)抑制剂治疗瘢痕性脱发:一项叙述性综述
    作者:
    Priya Agarwal, Oghenevoke Ajuchi, Tess M Lukowiak, Babar K Rao
    日期:
    2025-06-09

    Scarring alopecias, including lichen planopilaris, frontal fibrosing alopecia, folliculitis decalvans, central centrifugal cicatricial alopecia, discoid lupus erythematosus, and dissecting cellulitis cause permanent destruction of hair follicles, resulting in patches of hair loss that can be devastating for patients. Treatment options for scarring alopecias focus on disease stabilization and currently include corticosteroids and immunosuppressive agents, which often offer inconsistent disease improvements with waning patient satisfaction, especially in severe stages of the condition. Recent advances in therapeutics such as biologics and JAK inhibitors may offer some potential for disease stabilization and resolution through modulation of the inflammatory and immune-mediated pathways of scarring alopecias. This review examines literature reporting the off-label use of biologics and JAK inhibitors for the treatment of scarring alopecias. We find that TNF-α, IL-17, and JAK inhibitors demonstrate the most potential of currently available agents, with IL-23 and Interferon Alpha Receptor 1 inhibitors also showing some benefit.

  9. JCR分区: Q2 CAS分区: B4 影响因子: 2.5

    9. Wells syndrome: emerging triggers and treatments- an updated systematic review.

    作者:
    Areeba Ahmed, Brian Cahn, Roger Haber
    日期:
    2025-06-09

    Wells syndrome (eosinophilic cellulitis) is a rare inflammatory dermatosis characterized by erythematous, edematous plaques and dermal eosinophilic infiltration. Understanding its evolving triggers and treatment options is critical for optimizing management, particularly in corticosteroid-refractory cases. To systematically review newly reported immunologic and iatrogenic triggers of Wells syndrome, as well as emerging therapies, with the goal of updating clinical guidance. This review focuses on diagnosis and therapy, emphasizing outcomes in patients with refractory or relapsing disease. A systematic literature search was conducted following PRISMA 2020 guidelines across six databases for English-language studies published between January 2016 and May 2025. Studies were eligible if they described new triggers or treatments for Wells syndrome. Article selection and data extraction were performed independently by two reviewers. Risk of bias was assessed using the Joanna Briggs Institute and Newcastle-Ottawa tools. Twenty-four studies met inclusion criteria: 21 case reports, 2 case series, and 1 retrospective cohort study. Newly identified triggers included COVID-19 infection, SARS-CoV-2 and influenza vaccines, aluminum- and gelatin-containing pediatric vaccines, and biologic therapies such as ustekinumab and tumor necrosis factor-alpha (TNF-α) inhibitors. In vaccine-related cases, causality was supported by positive patch testing. Novel therapies trialed in corticosteroid-refractory or relapsing patients included dupilumab, topical ruxolitinib, abrocitinib, and mepolizumab. Most patients experienced complete or near-complete resolution. However, recurrences were common, particularly in idiopathic cases or upon re-exposure to known triggers. Recent literature expands the clinical spectrum of Wells syndrome, highlighting new immunologic and iatrogenic triggers. Targeted treatments, especially biologics and Janus kinase inhibitors, demonstrate promising results and may offer steroid-sparing alternatives for patients with refractory disease. Clinicians should consider emerging triggers in differential diagnosis and evaluate newer therapies in recurrent or treatment-resistant cases. Further prospective and registry-based studies are warranted to validate efficacy and support development of evidence-based management guidelines.

  10. JCR分区: Q2 CAS分区: B4 影响因子: 2.5

    10. Disproportionality analyses of Tapinarof-related adverse events based on the FAERS database.

    10. 基于FAERS数据库的Tapinarof相关不良事件的不成比例分析
    作者:
    Xia Huang, Congzhong Zhang, Lingjin Zhang
    日期:
    2025-06-09

    Tapinarof, a novel topical aryl hydrocarbon receptor (AhR) modulator, has demonstrated promising therapeutic efficacy in the treatment of plaque psoriasis. However, its comprehensive safety profile in real-world settings remains underexplored. To assess the post-marketing safety of Tapinarof using data from the FDA Adverse Event Reporting System (FAERS), with a focus on identifying significant adverse event (AE) signals and subgroup-specific risks. A retrospective disproportionality analysis was conducted on FAERS data from Q2 2022 to Q4 2024. Reports listing Tapinarof as the primary suspect drug were extracted and analyzed using four established signal detection algorithms (ROR, PRR, EBGM, IC), complemented by Bonferroni correction for multiple comparisons. A total of 1,227 AE reports were included. The majority of reports originated from adults aged 18-64 years and were submitted by healthcare professionals, with a balanced male-to-female ratio. The most frequently observed AE signals were related to skin and subcutaneous tissue disorders, including keratosis pilaris, contact dermatitis, and application site acne. Notable signals were also detected for infections (e.g., pustules) and hypersensitivity reactions. A novel tumor-related signal was identified for keratoacanthoma, with statistically significant associations also observed for squamous cell carcinoma of the skin. Gender subgroup analysis revealed a higher incidence of dermatological AEs in females and slightly stronger neoplasm-related signals in males. Age, weight, and indication data were largely missing, limiting subgroup-specific interpretation. Tapinarof is generally well tolerated in adults, particularly for psoriasis treatment, but is associated with strong cutaneous and immune-related AE signals. Novel findings-including pharyngeal swelling and tumor-related events-warrant heightened clinical vigilance, especially in patients requiring long-term or large-area application. Gender differences in AE reporting further underscore the need for personalized risk management strategies. Further longitudinal and mechanistic studies are essential to confirm these associations and guide safer clinical use.

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指标接近的期刊